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Blood changes with chronic illness and a theory about total and partial
wound units
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By
Dr Erik O. H. Enby, MD, Göteborg 1997.
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Abstract
Dr. Erik Enby has in the blood from chronically ill individuals
observed large amounts of unknown particles. The particles seem
to be able to invade the red blood cells and then these generally
show a strong tendency to get stuck to each other. The particles
are also involved in the construction of special structures that
are often visible in the blood and might cause somatic destruction
on various levels.
Purpose
Description of particles in blood from subjectively healthy people
and in connection with these particles structures growing in blood
from chronically ill people, and the presentation of a theory that
explains a change – or deterioration principle in the soma.
Method
Vital blood from thousands of chronically ill people belonging to
an average Swedish disease panorama has been studied in microscope,
with ordinary lightfield and with the Nomarski’s interference
contrast and it has been compared with vital blood from approximately
200 subjectively healthy individuals.
Result
Small oscillating 0,5 – 1-micrometer big particles were a
common finding in the blood from subjectively healthy people. In
the blood from chronically ill people, very often more, bigger and
variously shaped particles were found, that sometimes seemed to
invade the red blood cells. The particles also appeared in abundant
amounts together with a special structure principle that appeared
in different sizes among the blood cells.
Conclusion
Dr. Erik Enby suspects that some of the particles may cause formation
and growth of freely floating structures in the blood from chronically
ill individuals. The growth of these structures is assumed also
to take place in solid parts of the soma and thus cause local destruction
that either increase or heal with scars that will lead to either
stationary reduced or gradually decreased degree of efficiency of
the soma.
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Introduction
Darkfield microscoping of blood reveals that the blood in addition
to red and white blood cells contains large amounts of oscillating,
very tiny particles.
In the blood of chronically ill people several bigger particles,
sometimes of various shapes, occur. Many researchers have assumed
that these particles constitute some kind of microbial growth and
considered them as a possible cause of the development of chronic
disease conditions(1, 2). However,
the proof of this was never submitted to any analysis.
In order to find out the possible role of the particles in the development
of chronic disease conditions in general, the following microscopic
analysis was performed.
An ordinary lightfield microscoping 100 times enlarged was used
to get a first, quick orientation of the blood sample, and locally
a more special microscoping of this was carried out in co-operation
with the “Nomarski’s interference contrast method”.
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Material
and methods
The drop of blood was obtained from the fingertip. In order to avoid
any disturbance of the blood as far as possible, strong antiseptics
were avoided and the blood immediately could flow out into a thin
film between sterilized cover – and object glasses. The surface
of the blood film has an average of 13 cm2 and the microscoping
was performed immediately after the test.
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Microscoping
equipment
Leitz’ laboratory microscope Dialux 20, equipped with a 100W
halogen lamp. Modified UK condenser for darkfield, lightfield, phase
contrast and interference contrast. Plan-Fluotar objective. Binocular
photo tube FSA. All documentation has been compiled with Leitz’
fully automatic microscope camera Vario-Orthomat.
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Results
The blood cells in the blood from chronically ill people, different
from those of subjectively healthy people, in almost 100% of the
cases tended to get stuck to each other, and sometimes there were
no freely floating blood cells in the plasma (Figure
1).
Small spherical irregularly oscillating particles of the size of
up to one micrometer in diameter were often observed in the blood
plasma from subjectively healthy people. In the blood from chronically
ill people more and bigger particles often appeared. They appeared
single but also in small colonies in the plasma (Figure
2). Further they also seemed apt to be transformed into small,
longish forms, that appeared in both plasma and the red blood cells
that then usually were extremely changed in shape (Illustration
2)(3). In separate cases about 100% of the
red blood cells appeared in a thorn-apple form.
Round disc-shaped regions in the blood films were often observed
(Figure 3). Sometimes up to
5 of those appeared on an object glass (Illustration
1). The size of those varied up to 300 micrometer in diameter.
The inner part (nucleus) of a region like that was darker than
its
periphery (corona zone) and clearly demarcated against it (Illustration
1, illustration 2 and
figure 3)(4).
Interference contrast microscoping 1200 times enlarged, showed that
the darker inner part could be compared with an absolutely sterile
moon landscape, while the lighter periphery consisted of lots of
oscillating particles varying in shape, similar to those that could
be seen in the remaining blood. In some cases the occurrence of
these discoveries fluctuated from time to time and sometimes some
chronically ill people pervadingly showed a completely normal blood
morphology.
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Discussion
At the beginning of the examination there were three conditions in
the blood from chronically ill people that surprised:
a) The often most abundant numbers of oscillating particles of different
sizes and shapes occurred not only in the blood plasma, but also seemed
apt to invade the red blood cells.
b) The often most abundant numbers of deformed and apparently destroyed
blood cells.
c) The blood cells often tended to get stuck to each other. The question
arose if the deformation of and the agglutination tendency among them
were caused by the large amount of oscillating particles and thus
should be expected to occur already in the soma or if external influence
would be the reason therefore.
As the deformation of blood cells all the way into thorn-apple forms
sometimes could be almost 100% and the agglutination tendency so
strong that no free blood cells existed in the plasma and never
did so in the blood from subjectively healthy people, this was taken
as a grant that deformation and agglutination already existed in
the soma, and the suspicion that these phenomena only were artefacts,
was reduced.
Since the agglutination occurs even in other body fluids, for example
when lactic acid bacteria sour the milk, by analogy therewith a
powerful lumping of the red blood cells could be an infection symptom.
Thus, the agglutination tendency of the red blood cells in the blood
from chronically ill people may indicate a spread microbial growth
in the body fluids by analogy with an infection.
This made one look upon the often occurring large amount of particles
in the blood plasma and blood cells in the blood from chronically
ill people as microorganisms. Might it be that sometimes these may
also attack the blood cells and thus change their form and that
the agglutination tendency came into existence because a microbial
growth existed in the blood?(5)
Consequently it is noticeable that these three conditions often
occurred in the blood of chronically ill people and therefore this
was taken as a proof for an ongoing development of a disturbance
in the soma. Since also blood from subjectively healthy people occasionally
changed in a similar way the question arose, would this indicate
a beginning somatic disturbance when the individual was still in
the so-called period of incubation. So far the microscoping had
only given a hint of all this. It was mostly the vertical source
of knowledge, that is the intuition, in co-operation with my own
experience, that had to be the guide in order to decide if a blood
picture seemed to be disturbed or not(6).
As agglutination tendency and deformed blood cells hardly existed
with subjectively healthy people, it was supposed that the blood
from a chronically ill person, due to increased viscosity and the
often large amount of bad blood cells, probably serves its functions
in the soma with a lower degree of efficiency than is the case with
subjectively healthy people, whose blood cells were lying freely
and had a normal look.
Not until it became apparent that the particles sometimes formed
conglomerations in the blood plasma and existed in large amounts
in the corona zones of the disc-shaped regions, it seemed possible
to get an idea of their importance on a more rational level. It
couldn’t be wrong to consider these regions a result of a
flattening of spherical structures floating among the blood cells
in the drop of blood that floated out between the object –
and cover glasses and thus formed a transparent, thin blood film.
Further, you could imagine that these structures are limited against
surrounding blood by a layer consisting of the particles visible
in the corona zones (Illustration
3).
Since these postulated spherical structures must have been formed
and the regions have different sizes, it was assumed that they grow
and that the peripheral particle layer makes the growth zone. The
increasing size of the structures would later on mean that the capillary
net will catch them, which would be the reason why they are often
found in drops of blood from for example the fingertip.
These floating, postulating structures among the blood cells that
for easily explained reasons still haven’t been observed in
their natural state, of course, do exist and they will now be the
base for the continued discussion. This will deal with the possibility
that solid tissues may form substratum for this type of growing
structures with a particle front to the surroundings (Illustration
4, illustration 5).
On the spot for the growth of structure, tissue destruction then
would take place and sometimes a perforation of the organ surface
with outwards emptying of the structure contents will take place
(Illustration 6). This
is generally called pus and is then accordingly to this theory equivalent
to the darker part, nucleus, in a disc-shaped region.
The defect that occurred on the organ surface can be classified
as a wound and since the postulating growing structures actually
can lead to tissue destruction and wound, hereafter they will be
called wound units.
A wound unit in a solid tissue would before perforation outwards be
the total so-called internal wound, the spherical wound surface bordering
the surrounding tissue holds the wound contents. At perforation the
total internal wound is changed into a partially external wound, whose
bowl-shaped wound surface will be covered with rests from the structure
contents and in size be equivalent to only part of the theoretically
totally possible wound surface. In both cases, of course, there is
a particle front or growth zone to an adjacent tissue (Illustration
7).
The cells that have made room for inner and outer wound formations
in the blood may have their equivalence in the devitalized blood
cells changed in shape, which so often occur in connection with
chronic disease.
Wound units might lead to tissue destruction in any part of the
soma. The local tissue losses certainly in most cases are replaced
by a stationary scar tissue, but sometimes this might not occur.
In the latter case, the wound stays open and if the particle front
against surrounding tissue stays active, it can also increase in
size. As something in between, an increasingly widespread scar formation
may be a possibility if the scar tissue is formed directly on the
track of the particle front. The different alternatives will lead
to either a stationary reduced or in various respects a gradual
decrease of efficiency with the soma.
The fact is that blood from chronically ill people can be completely
free from the above-mentioned findings and look perfectly normal.
This may indicate an inherent ability on the particles to be drawn
to and stay in other areas of the soma.
The fact that they seem to be able to transform into small, longish
forms may mean that they also change characteristics and look for
other environments than the one offered by the blood. Another possibility
is that they have been buried among agglutinated red blood cells,
meaning that they are taking part in the agglutination. This might
be some explanations of the blood sometimes being completely free
from the above described findings, even if a severe chronic disease
exists.
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Epilogue
In the plasma among the blood cells in the blood from chronically
ill people, lots of variously formed particles exist. They can exist
separately or form colonies and they can also invade the red blood
cells. Probably several different types of particles exist. A common
characteristic of these is that they all seem to be involved in
the growing process of architecturally similar structures, some
of which are probably able to grow in solid tissues as well.
In accordance with a dominating growth process changing and finally
“impoverishing” a ground, one may believe that such
processes in the soma slowly devastate it and in that way different
disease conditions may be realized. A first knowledge of such growing
processes can be gained by blood microscoping.
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References
(1) Béchamp Antoine
(1911). The blood and its third anatomical
element. Philadelphia. Boericke & Tafel.
(2) Enderlein, Günther
(1981). Bakterien-Cyclogenie. (2.
Ausgabe). Hoya. Semmelweis-Verlag.
(3) Enby Erik O. H.
(1989). Die Präsenz zyklischer mikrobischer
Prozesse Nachgewiesen im Blut von chronisch erkrankten Patienten.
(The
presence of cyclical microbial processes indicated in the blood
of patients with chronic diseases). Hoya. Semmelweis-Verlag.
(4) Enby, Erik O. H.
(1984). Mikrobliknande bildningar i blod
vid kroniska sjukdomar. (Microbe-like
formations in the blood of chronically diseased individuals).
Svensk Tidskrift för Biologisk Medicin, Swedish Journal of
Biological Medicine. No 1. p 22-26.
(5) Scholander C., Treutiger, C.J., Hultenby, K.
& Wahlgren, M.
(1996). Novel fabrillar structure confers
adhesive property to malaria-infected erythrocytes. Nature
Medicine. Nr 2. s 204-208.
(6) Johannes Paulus II
(1992). Vetenskapens världsbild och
tron. Signum. Volume 18. No 9/10. p 301-304.
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© 1997-2004. Dr Erik Enby. All rights reserved. This article
may only be reproduced in its entirety.
Illustrations: Lisa Örtengren.
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